Oxidative Stress
Corrigendum to "Adipose stem cells-derived microvesicles and chicken egg-derived exosomes attenuate cardiac ischemia/reperfusion injury through AKT/ERK/Nrf2/HO-1 axis to inhibit apoptosis and inflammation and restore autophagy" [Life Sci. 395 (2026) 124364].
Life sciences 2026-Jul-03
Huang Chi-Ming, Cheng Yu-Hsuan, Lin Yi-Hua, Chien Chiang-Ting
Abstract
Key Findings
- Adipose stem cells-derived microvesicles and chicken egg-derived exosomes attenuate cardiac ischemia/reperfusion injury.
- The protective effect is mediated through the AKT/ERK/Nrf2/HO-1 signaling axis.
- This mechanism inhibits apoptosis and inflammation while restoring autophagy in cardiac tissue.
Clinical Significance
These findings suggest a potential therapeutic strategy to reduce cardiac damage following ischemia/reperfusion injury by targeting oxidative stress pathways and enhancing cellular repair mechanisms.
Citation
Huang Chi-Ming, Cheng Yu-Hsuan, Lin Yi-Huaet al.. Corrigendum to "Adipose stem cells-derived microvesicles and chicken egg-derived exosomes attenuate cardiac ischemia/reperfusion injury through AKT/ERK/Nrf2/HO-1 axis to inhibit apoptosis and inflammation and restore autophagy" [Life Sci. 395 (2026) 124364]. Life sciences. 2026-Jul-03.
DOI: 10.1016/j.lfs.2026.124561