Oxidative Stress

Corrigendum to "Adipose stem cells-derived microvesicles and chicken egg-derived exosomes attenuate cardiac ischemia/reperfusion injury through AKT/ERK/Nrf2/HO-1 axis to inhibit apoptosis and inflammation and restore autophagy" [Life Sci. 395 (2026) 124364].

Life sciences

Abstract

Key Findings

  • Adipose stem cells-derived microvesicles and chicken egg-derived exosomes attenuate cardiac ischemia/reperfusion injury.
  • The protective effect is mediated through the AKT/ERK/Nrf2/HO-1 signaling axis.
  • This mechanism inhibits apoptosis and inflammation while restoring autophagy in cardiac tissue.

Clinical Significance

These findings suggest a potential therapeutic strategy to reduce cardiac damage following ischemia/reperfusion injury by targeting oxidative stress pathways and enhancing cellular repair mechanisms.

Citation

Huang Chi-Ming, Cheng Yu-Hsuan, Lin Yi-Huaet al.. Corrigendum to "Adipose stem cells-derived microvesicles and chicken egg-derived exosomes attenuate cardiac ischemia/reperfusion injury through AKT/ERK/Nrf2/HO-1 axis to inhibit apoptosis and inflammation and restore autophagy" [Life Sci. 395 (2026) 124364]. Life sciences. 2026-Jul-03.

DOI: 10.1016/j.lfs.2026.124561