Ferroptosis

Corrigendum to "Enhancing myocardial injury recovery: Nrf2 activation by sulforaphane regulates ferroptosis and oxidative stress in takotsubo-like models" [Int. Immunopharmacol. 167 (2025) 115659].

International immunopharmacology

Abstract

Key Findings

  • Nrf2 activation by sulforaphane enhances recovery from myocardial injury
  • Nrf2 regulates ferroptosis in takotsubo-like cardiac injury models
  • Sulforaphane reduces oxidative stress contributing to myocardial protection

Clinical Significance

Targeting Nrf2 activation with sulforaphane may offer a therapeutic approach to mitigate myocardial injury by modulating ferroptosis and oxidative stress, potentially improving outcomes in takotsubo cardiomyopathy.

Citation

Huang Yuxi, Tang Wenyi, Xu Meiet al.. Corrigendum to "Enhancing myocardial injury recovery: Nrf2 activation by sulforaphane regulates ferroptosis and oxidative stress in takotsubo-like models" [Int. Immunopharmacol. 167 (2025) 115659]. International immunopharmacology. 2026-Aug-29.

DOI: 10.1016/j.intimp.2026.117319